Collagen Deposition & Biasing
Session Objective
To differentiate between temporary Edema (vascular swelling) and permanent Collagen Accretion. We analyze how to bias mechanical stress to favor specific vectors—longitudinal or circumferential expansion—by targeting the layers of the Tunica Albuginea.
1. Positive Tissue Accretion
Permanent growth is the result of successful integration of new Extracellular Matrix (ECM). When mechanotransduction triggers the fibroblasts, they secrete pro-collagen, which must be woven into the existing structural lattice through a process called fibrillogenesis.
Fibrillogenesis
The biological assembly of collagen fibrils. This process determines the density and "permanence" of the new tissue mass. It requires the building blocks from Module 02.
Cross-Linking
The formation of chemical bonds between collagen strands. This is the "curing" process that prevents the tissue from recoiling to its original size during de-loading.
2. Biasing: Length vs. Girth
The Tunica Albuginea is a bi-layered structure. By manipulating the Vector of Force, we can dictate which layer undergoes the most significant remodeling. Length requires axial tension, while girth requires radial expansion.
| Growth Vector | Mechanical Bias | Biological Target |
|---|---|---|
| Length | Axial Tension (Traction) | Outer Longitudinal Layer |
| Girth | Radial Expansion (Pressure) | Inner Circular Layer |
3. Verification: Edema vs. Growth
It is vital to distinguish between Interstitial Fluid (temporary swelling) and Cellular Hyperplasia (permanent growth). We use the Tissue Density Audit to ensure the gains recorded are structural rather than inflammatory.
The "Permanence" Check
- Rest Requirement 48 Hours
- Tissue Palpation Dense / Firm
- Retraction Test Minimal Recoil